Arsenic induces apoptosis of multidrug-resistant human myeloid leukemia cells that express Bcr-Abl or overexpress MDR, MRP, Bcl-2, or Bcl-x(L). [electronic resource]
Producer: 20000224Description: 1014-22 p. digitalISSN:- 0006-4971
- ATP Binding Cassette Transporter, Subfamily B, Member 1 -- biosynthesis
- ATP-Binding Cassette Transporters -- biosynthesis
- Acetylation -- drug effects
- Antineoplastic Agents -- pharmacology
- Apoptosis -- drug effects
- Apoptotic Protease-Activating Factor 1
- Arsenic Trioxide
- Arsenicals -- pharmacology
- Caspases -- metabolism
- Cytochrome c Group -- metabolism
- Cytosol -- metabolism
- Drug Resistance, Multiple
- Drug Resistance, Neoplasm
- Fas Ligand Protein
- Fusion Proteins, bcr-abl -- biosynthesis
- Gene Expression Regulation, Leukemic
- Genes, bcl-2
- HL-60 Cells -- drug effects
- Histones -- metabolism
- Humans
- Immunophenotyping
- K562 Cells -- drug effects
- Leukemia, Myelogenous, Chronic, BCR-ABL Positive -- genetics
- Macrophage-1 Antigen -- biosynthesis
- Membrane Glycoproteins -- biosynthesis
- Mitochondria -- metabolism
- Multidrug Resistance-Associated Proteins
- Neoplasm Proteins -- biosynthesis
- Oxides -- pharmacology
- Protein Biosynthesis
- Protein Processing, Post-Translational -- drug effects
- Proteins -- genetics
- Proto-Oncogene Proteins -- biosynthesis
- Proto-Oncogene Proteins c-bcl-2 -- biosynthesis
- bcl-2-Associated X Protein
- bcl-X Protein
- fas Receptor -- biosynthesis
No physical items for this record
Publication Type: Journal Article
There are no comments on this title.
Log in to your account to post a comment.