Targeted deficiency or cytosolic truncation of the VE-cadherin gene in mice impairs VEGF-mediated endothelial survival and angiogenesis. [electronic resource]
Producer: 19990813Description: 147-57 p. digitalISSN:- 0092-8674
- Animals
- Antigens, CD
- Apoptosis -- physiology
- Cadherins -- genetics
- Cell Survival -- physiology
- Cytoskeletal Proteins -- physiology
- Cytosol -- chemistry
- DNA Primers
- Endothelial Growth Factors -- physiology
- Endothelium, Vascular -- chemistry
- Fetus -- cytology
- Gene Expression Regulation, Developmental
- Hematopoiesis -- physiology
- In Situ Nick-End Labeling
- Intercellular Junctions -- physiology
- Lymphokines -- physiology
- Mice
- Mice, Transgenic
- Microscopy, Electron
- Mutagenesis, Site-Directed
- Neovascularization, Physiologic -- physiology
- Phosphatidylinositol 3-Kinases -- metabolism
- Receptor Protein-Tyrosine Kinases -- physiology
- Receptors, Growth Factor -- physiology
- Receptors, Vascular Endothelial Growth Factor
- Signal Transduction -- physiology
- Trans-Activators
- Vascular Endothelial Growth Factor A
- Vascular Endothelial Growth Factors
- beta Catenin
No physical items for this record
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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