Induction of tenascin-C in cardiac myocytes by mechanical deformation. Role of reactive oxygen species. [electronic resource]
Producer: 19990819Description: 21840-6 p. digitalISSN:- 0021-9258
- 1,2-Dihydroxybenzene-3,5-Disulfonic Acid Disodium Salt -- pharmacology
- Acetylcysteine -- pharmacology
- Alternative Splicing
- Angiotensin Receptor Antagonists
- Animals
- Animals, Newborn
- Antioxidants -- pharmacology
- Catalase -- pharmacology
- Cells, Cultured
- DNA-Binding Proteins -- genetics
- Fibronectins -- genetics
- Gene Expression Regulation -- drug effects
- Genes, Reporter
- Heart Ventricles
- I-kappa B Kinase
- I-kappa B Proteins
- Indazoles -- pharmacology
- Myocardium -- cytology
- NF-KappaB Inhibitor alpha
- Osteonectin -- genetics
- Promoter Regions, Genetic
- Protein Isoforms -- genetics
- Protein Serine-Threonine Kinases -- genetics
- RNA, Messenger -- genetics
- Rats
- Rats, Sprague-Dawley
- Reactive Oxygen Species -- metabolism
- Receptor, Angiotensin, Type 1
- Receptor, Angiotensin, Type 2
- Stress, Mechanical
- Tenascin -- biosynthesis
- Transcription, Genetic -- drug effects
- Transfection
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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