Induction of tenascin-C in cardiac myocytes by mechanical deformation. Role of reactive oxygen species.

Yamamoto, K

Induction of tenascin-C in cardiac myocytes by mechanical deformation. Role of reactive oxygen species. [electronic resource] - The Journal of biological chemistry Jul 1999 - 21840-6 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.

0021-9258

10.1074/jbc.274.31.21840 doi


1,2-Dihydroxybenzene-3,5-Disulfonic Acid Disodium Salt--pharmacology
Acetylcysteine--pharmacology
Alternative Splicing
Angiotensin Receptor Antagonists
Animals
Animals, Newborn
Antioxidants--pharmacology
Catalase--pharmacology
Cells, Cultured
DNA-Binding Proteins--genetics
Fibronectins--genetics
Gene Expression Regulation--drug effects
Genes, Reporter
Heart Ventricles
I-kappa B Kinase
I-kappa B Proteins
Indazoles--pharmacology
Myocardium--cytology
NF-KappaB Inhibitor alpha
Osteonectin--genetics
Promoter Regions, Genetic
Protein Isoforms--genetics
Protein Serine-Threonine Kinases--genetics
RNA, Messenger--genetics
Rats
Rats, Sprague-Dawley
Reactive Oxygen Species--metabolism
Receptor, Angiotensin, Type 1
Receptor, Angiotensin, Type 2
Stress, Mechanical
Tenascin--biosynthesis
Transcription, Genetic--drug effects
Transfection