Suppression of PTPN6 exacerbates aluminum oxide nanoparticle-induced COPD-like lesions in mice through activation of STAT pathway.
Li, Xiaobo
Suppression of PTPN6 exacerbates aluminum oxide nanoparticle-induced COPD-like lesions in mice through activation of STAT pathway. [electronic resource] - Particle and fibre toxicology Dec 2017 - 53 p. digital
Publication Type: Journal Article
1743-8977
10.1186/s12989-017-0234-0 doi
A549 Cells
Aluminum Oxide--toxicity
Animals
Apoptosis--drug effects
Apoptosis Regulatory Proteins--metabolism
Disease Progression
Dose-Response Relationship, Drug
Humans
Inflammation Mediators--metabolism
Inhalation Exposure--adverse effects
Lung--drug effects
Male
Metal Nanoparticles--toxicity
Mice, Inbred C57BL
Phosphorylation
Pneumonia--chemically induced
Protein Tyrosine Phosphatase, Non-Receptor Type 6--metabolism
Pulmonary Disease, Chronic Obstructive--chemically induced
Pulmonary Emphysema--chemically induced
RNA-Binding Proteins--metabolism
STAT3 Transcription Factor--metabolism
Signal Transduction--drug effects
Time Factors
Suppression of PTPN6 exacerbates aluminum oxide nanoparticle-induced COPD-like lesions in mice through activation of STAT pathway. [electronic resource] - Particle and fibre toxicology Dec 2017 - 53 p. digital
Publication Type: Journal Article
1743-8977
10.1186/s12989-017-0234-0 doi
A549 Cells
Aluminum Oxide--toxicity
Animals
Apoptosis--drug effects
Apoptosis Regulatory Proteins--metabolism
Disease Progression
Dose-Response Relationship, Drug
Humans
Inflammation Mediators--metabolism
Inhalation Exposure--adverse effects
Lung--drug effects
Male
Metal Nanoparticles--toxicity
Mice, Inbred C57BL
Phosphorylation
Pneumonia--chemically induced
Protein Tyrosine Phosphatase, Non-Receptor Type 6--metabolism
Pulmonary Disease, Chronic Obstructive--chemically induced
Pulmonary Emphysema--chemically induced
RNA-Binding Proteins--metabolism
STAT3 Transcription Factor--metabolism
Signal Transduction--drug effects
Time Factors