SIRT1-mediated deacetylation of PGC1α attributes to the protection of curcumin against glutamate excitotoxicity in cortical neurons.

Jia, Ning

SIRT1-mediated deacetylation of PGC1α attributes to the protection of curcumin against glutamate excitotoxicity in cortical neurons. [electronic resource] - Biochemical and biophysical research communications 09 2016 - 1376-81 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't

1090-2104

10.1016/j.bbrc.2016.08.132 doi


Acetylation--drug effects
Adenosine Triphosphate--metabolism
Animals
Benzamides--pharmacology
Calcium--metabolism
Cell Death--drug effects
Cells, Cultured
Cerebral Cortex--pathology
Curcumin--pharmacology
Electron Transport Complex IV--metabolism
Glutamic Acid--toxicity
Intracellular Space--metabolism
Membrane Potential, Mitochondrial--drug effects
Mitochondria--drug effects
Naphthols--pharmacology
Neurons--drug effects
Neuroprotection--drug effects
Neuroprotective Agents--pharmacology
Neurotoxins--toxicity
Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha--metabolism
Rats, Sprague-Dawley
Reactive Oxygen Species--metabolism
Sirtuin 1--metabolism