Insights into the molecular mechanism for type 2 diabetes susceptibility at the KCNQ1 locus from temporal changes in imprinting status in human islets.

Travers, Mary E

Insights into the molecular mechanism for type 2 diabetes susceptibility at the KCNQ1 locus from temporal changes in imprinting status in human islets. [electronic resource] - Diabetes Mar 2013 - 987-92 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't

1939-327X

10.2337/db12-0819 doi


Adult
Alleles
Chromosomes, Human, Pair 11--genetics
Cohort Studies
Cyclin-Dependent Kinase Inhibitor p57--genetics
DNA Methylation
Diabetes Mellitus, Type 2--genetics
Fetal Development
Gene Expression Regulation, Developmental
Genetic Association Studies
Genetic Loci
Genetic Predisposition to Disease
Humans
Introns
Islets of Langerhans--growth & development
KCNQ1 Potassium Channel--genetics
Pancreas--embryology
Polymorphism, Single Nucleotide
Potassium Channels, Voltage-Gated--genetics
United Kingdom