Cellular growth inhibition by FK778 is linked to G1 arrest or S phase accumulation, dependent on the functional status of the retinoblastoma protein.
Hoppe-Seyler, Karin
Cellular growth inhibition by FK778 is linked to G1 arrest or S phase accumulation, dependent on the functional status of the retinoblastoma protein. [electronic resource] - International journal of molecular medicine Mar 2009 - 415-20 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1107-3756
10.3892/ijmm_00000146 doi
Alkynes--pharmacology
Antiviral Agents--pharmacology
DNA-Binding Proteins--biosynthesis
G1 Phase--drug effects
HeLa Cells
Human papillomavirus 16--metabolism
Human papillomavirus 18--metabolism
Humans
Isoxazoles--pharmacology
Nitriles--pharmacology
Oncogene Proteins, Viral--biosynthesis
Papillomavirus E7 Proteins
Phosphorylation--drug effects
Retinoblastoma Protein--metabolism
S Phase--drug effects
Tumor Suppressor Protein p53--metabolism
Cellular growth inhibition by FK778 is linked to G1 arrest or S phase accumulation, dependent on the functional status of the retinoblastoma protein. [electronic resource] - International journal of molecular medicine Mar 2009 - 415-20 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1107-3756
10.3892/ijmm_00000146 doi
Alkynes--pharmacology
Antiviral Agents--pharmacology
DNA-Binding Proteins--biosynthesis
G1 Phase--drug effects
HeLa Cells
Human papillomavirus 16--metabolism
Human papillomavirus 18--metabolism
Humans
Isoxazoles--pharmacology
Nitriles--pharmacology
Oncogene Proteins, Viral--biosynthesis
Papillomavirus E7 Proteins
Phosphorylation--drug effects
Retinoblastoma Protein--metabolism
S Phase--drug effects
Tumor Suppressor Protein p53--metabolism