Type 1 Immune Mechanisms Driven by the Response to Infection with Attenuated Rabies Virus Result in Changes in the Immune Bias of the Tumor Microenvironment and Necrosis of Mouse GL261 Brain Tumors. [electronic resource]
Producer: 20170922Description: 4513-4523 p. digitalISSN:- 1550-6606
- Animals
- B-Lymphocytes -- immunology
- Blood-Brain Barrier -- immunology
- Brain -- immunology
- Brain Neoplasms -- immunology
- CD4-Positive T-Lymphocytes
- Cytokines -- immunology
- Disease Models, Animal
- Female
- Interferon-gamma -- biosynthesis
- Mice
- Necrosis -- virology
- Nitric Oxide Synthase Type II -- biosynthesis
- Rabies virus -- genetics
- T-Box Domain Proteins -- deficiency
- Th2 Cells -- immunology
- Tumor Microenvironment -- immunology
- Tumor Necrosis Factor-alpha -- biosynthesis
- T-bet Transcription Factor
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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