Type 1 Immune Mechanisms Driven by the Response to Infection with Attenuated Rabies Virus Result in Changes in the Immune Bias of the Tumor Microenvironment and Necrosis of Mouse GL261 Brain Tumors.

Bongiorno, Emily K

Type 1 Immune Mechanisms Driven by the Response to Infection with Attenuated Rabies Virus Result in Changes in the Immune Bias of the Tumor Microenvironment and Necrosis of Mouse GL261 Brain Tumors. [electronic resource] - Journal of immunology (Baltimore, Md. : 1950) 06 2017 - 4513-4523 p. digital

Publication Type: Journal Article; Research Support, N.I.H., Extramural

1550-6606

10.4049/jimmunol.1601444 doi


Animals
B-Lymphocytes--immunology
Blood-Brain Barrier--immunology
Brain--immunology
Brain Neoplasms--immunology
CD4-Positive T-Lymphocytes
Cytokines--immunology
Disease Models, Animal
Female
Interferon-gamma--biosynthesis
Mice
Necrosis--virology
Nitric Oxide Synthase Type II--biosynthesis
Rabies virus--genetics
T-Box Domain Proteins--deficiency
Th2 Cells--immunology
Tumor Microenvironment--immunology
Tumor Necrosis Factor-alpha--biosynthesis
T-bet Transcription Factor