Chronic GluN2B antagonism disrupts behavior in wild-type mice without protecting against synapse loss or memory impairment in Alzheimer's disease mouse models. [electronic resource]
Producer: 20140804Description: 8277-88 p. digitalISSN:- 1529-2401
- Alzheimer Disease -- complications
- Amyloid beta-Peptides -- metabolism
- Amyloid beta-Protein Precursor -- genetics
- Animals
- Attention Deficit and Disruptive Behavior Disorders -- chemically induced
- Behavior, Animal -- drug effects
- Disease Models, Animal
- Female
- HEK293 Cells
- Humans
- In Vitro Techniques
- Male
- Maze Learning -- drug effects
- Membrane Potentials -- drug effects
- Memory Disorders -- etiology
- Mice
- Mice, Inbred C57BL
- Mice, Transgenic
- Mutation -- genetics
- Piperidines -- pharmacology
- Synapses -- pathology
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Publication Type: Journal Article
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