Transactivation of epidermal growth factor receptor by enhanced levels of endogenous angiotensin II contributes to the overexpression of Giα proteins in vascular smooth muscle cells from SHR. [electronic resource]
Producer: 20111230Description: 1716-26 p. digitalISSN:- 1873-3913
- Acetylcysteine -- pharmacology
- Adenylyl Cyclases -- metabolism
- Angiotensin II -- genetics
- Angiotensin-Converting Enzyme Inhibitors -- pharmacology
- Animals
- CSK Tyrosine-Protein Kinase
- Colforsin -- pharmacology
- ErbB Receptors -- genetics
- GTP-Binding Protein alpha Subunits, Gi-Go -- genetics
- Gene Expression -- drug effects
- Guanosine 5'-O-(3-Thiotriphosphate) -- pharmacology
- Humans
- Hypertension -- genetics
- Mitogen-Activated Protein Kinases -- genetics
- Muscle, Smooth, Vascular -- cytology
- Myocytes, Smooth Muscle -- cytology
- Oxidative Stress -- drug effects
- Phosphorylation
- Protein-Tyrosine Kinases -- genetics
- Rats
- Rats, Inbred SHR
- Rats, Inbred WKY
- Signal Transduction -- drug effects
- Transcriptional Activation -- drug effects
- src-Family Kinases
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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