Electrophilic components of diesel exhaust particles (DEP) activate transient receptor potential ankyrin-1 (TRPA1): a probable mechanism of acute pulmonary toxicity for DEP. [electronic resource]
Producer: 20111014Description: 950-9 p. digitalISSN:- 1520-5010
- Animals
- Calcium Channels -- genetics
- Carbon -- toxicity
- Cell Line
- Cells, Cultured
- Coal Ash
- Ganglia, Spinal -- cytology
- Humans
- Lung Diseases -- chemically induced
- Mice
- Mice, Inbred C57BL
- Mutation
- Nerve Tissue Proteins -- agonists
- Neurons -- drug effects
- Particulate Matter -- toxicity
- TRPA1 Cation Channel
- Transient Receptor Potential Channels -- agonists
- Vehicle Emissions -- toxicity
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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