Electrophilic components of diesel exhaust particles (DEP) activate transient receptor potential ankyrin-1 (TRPA1): a probable mechanism of acute pulmonary toxicity for DEP.
Deering-Rice, Cassandra E
Electrophilic components of diesel exhaust particles (DEP) activate transient receptor potential ankyrin-1 (TRPA1): a probable mechanism of acute pulmonary toxicity for DEP. [electronic resource] - Chemical research in toxicology Jun 2011 - 950-9 p. digital
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
1520-5010
10.1021/tx200123z doi
Animals
Calcium Channels--genetics
Carbon--toxicity
Cell Line
Cells, Cultured
Coal Ash
Ganglia, Spinal--cytology
Humans
Lung Diseases--chemically induced
Mice
Mice, Inbred C57BL
Mutation
Nerve Tissue Proteins--agonists
Neurons--drug effects
Particulate Matter--toxicity
TRPA1 Cation Channel
Transient Receptor Potential Channels--agonists
Vehicle Emissions--toxicity
Electrophilic components of diesel exhaust particles (DEP) activate transient receptor potential ankyrin-1 (TRPA1): a probable mechanism of acute pulmonary toxicity for DEP. [electronic resource] - Chemical research in toxicology Jun 2011 - 950-9 p. digital
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
1520-5010
10.1021/tx200123z doi
Animals
Calcium Channels--genetics
Carbon--toxicity
Cell Line
Cells, Cultured
Coal Ash
Ganglia, Spinal--cytology
Humans
Lung Diseases--chemically induced
Mice
Mice, Inbred C57BL
Mutation
Nerve Tissue Proteins--agonists
Neurons--drug effects
Particulate Matter--toxicity
TRPA1 Cation Channel
Transient Receptor Potential Channels--agonists
Vehicle Emissions--toxicity