HDAC1/NFκB pathway is involved in curcumin inhibiting of Tat-mediated long terminal repeat transactivation. [electronic resource]
Producer: 20111114Description: 3385-91 p. digitalISSN:- 1097-4652
- AMP-Activated Protein Kinases -- metabolism
- Acetyl-CoA Carboxylase -- metabolism
- Acetylation
- Anti-HIV Agents -- pharmacology
- Binding Sites
- Chromatin Assembly and Disassembly -- drug effects
- Curcumin -- pharmacology
- Dose-Response Relationship, Drug
- Down-Regulation
- Energy Metabolism -- drug effects
- Enzyme Activation
- HIV Long Terminal Repeat -- drug effects
- HIV-1 -- drug effects
- HeLa Cells
- Histone Deacetylase 1 -- genetics
- Histones -- metabolism
- Humans
- I-kappa B Kinase -- metabolism
- Phosphorylation
- RNA Interference
- Signal Transduction -- drug effects
- Transcription Factor RelA -- metabolism
- Transcriptional Activation -- drug effects
- Transfection
- p300-CBP Transcription Factors -- metabolism
- tat Gene Products, Human Immunodeficiency Virus -- genetics
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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