HDAC1/NFκB pathway is involved in curcumin inhibiting of Tat-mediated long terminal repeat transactivation.

Zhang, Hong-Sheng

HDAC1/NFκB pathway is involved in curcumin inhibiting of Tat-mediated long terminal repeat transactivation. [electronic resource] - Journal of cellular physiology Dec 2011 - 3385-91 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't

1097-4652

10.1002/jcp.22691 doi


AMP-Activated Protein Kinases--metabolism
Acetyl-CoA Carboxylase--metabolism
Acetylation
Anti-HIV Agents--pharmacology
Binding Sites
Chromatin Assembly and Disassembly--drug effects
Curcumin--pharmacology
Dose-Response Relationship, Drug
Down-Regulation
Energy Metabolism--drug effects
Enzyme Activation
HIV Long Terminal Repeat--drug effects
HIV-1--drug effects
HeLa Cells
Histone Deacetylase 1--genetics
Histones--metabolism
Humans
I-kappa B Kinase--metabolism
Phosphorylation
RNA Interference
Signal Transduction--drug effects
Transcription Factor RelA--metabolism
Transcriptional Activation--drug effects
Transfection
p300-CBP Transcription Factors--metabolism
tat Gene Products, Human Immunodeficiency Virus--genetics