Stimulation of FasL induces production of proinflammatory mediators through activation of mitogen-activated protein kinases and nuclear factor-κB in THP-1 cells. [electronic resource]
Producer: 20120731Description: 1-10 p. digitalISSN:- 1573-2576
- Antibodies, Monoclonal
- Antigens, Surface -- immunology
- Cell Line, Tumor
- Extracellular Signal-Regulated MAP Kinases -- metabolism
- Fas Ligand Protein -- genetics
- Humans
- Interleukin-8 -- biosynthesis
- JNK Mitogen-Activated Protein Kinases -- metabolism
- MAP Kinase Signaling System
- Macrophage Activation
- Macrophages -- immunology
- Matrix Metalloproteinase 9 -- biosynthesis
- Membrane Glycoproteins -- immunology
- NF-kappa B -- metabolism
- RNA Interference
- RNA, Small Interfering
- Receptors, Fc -- genetics
- Recombinant Fusion Proteins -- pharmacology
- Tumor Necrosis Factor-alpha -- biosynthesis
- fas Receptor -- genetics
- p38 Mitogen-Activated Protein Kinases -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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