Stimulation of FasL induces production of proinflammatory mediators through activation of mitogen-activated protein kinases and nuclear factor-κB in THP-1 cells.

Lee, Sang-Min

Stimulation of FasL induces production of proinflammatory mediators through activation of mitogen-activated protein kinases and nuclear factor-κB in THP-1 cells. [electronic resource] - Inflammation Feb 2012 - 1-10 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't

1573-2576

10.1007/s10753-010-9283-3 doi


Antibodies, Monoclonal
Antigens, Surface--immunology
Cell Line, Tumor
Extracellular Signal-Regulated MAP Kinases--metabolism
Fas Ligand Protein--genetics
Humans
Interleukin-8--biosynthesis
JNK Mitogen-Activated Protein Kinases--metabolism
MAP Kinase Signaling System
Macrophage Activation
Macrophages--immunology
Matrix Metalloproteinase 9--biosynthesis
Membrane Glycoproteins--immunology
NF-kappa B--metabolism
RNA Interference
RNA, Small Interfering
Receptors, Fc--genetics
Recombinant Fusion Proteins--pharmacology
Tumor Necrosis Factor-alpha--biosynthesis
fas Receptor--genetics
p38 Mitogen-Activated Protein Kinases--metabolism