Abnormal modulation of cell protective systems in response to ischemic/reperfusion injury is important in the development of mouse sickle cell hepatopathy. [electronic resource]
Producer: 20110526Description: 24-32 p. digitalISSN:- 1592-8721
- Anemia, Sickle Cell -- etiology
- Animals
- Blotting, Western
- Cells, Cultured
- Cytoprotection
- Female
- HSP27 Heat-Shock Proteins -- genetics
- HSP70 Heat-Shock Proteins -- genetics
- Heme Oxygenase-1 -- genetics
- Hepatocytes -- cytology
- Humans
- Liver Diseases -- etiology
- Male
- Mice
- Mice, Inbred C57BL
- Mice, Inbred Strains
- Mice, Transgenic
- NF-kappa B -- genetics
- Nitric Oxide -- metabolism
- Nitric Oxide Synthase Type II -- genetics
- Nitric Oxide Synthase Type III -- genetics
- Peroxiredoxins -- genetics
- RNA, Messenger -- genetics
- Reperfusion Injury -- complications
- Reverse Transcriptase Polymerase Chain Reaction
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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