Cyclin-dependent kinase inhibitors and JNK act as molecular switches, regulating the choice between growth arrest and apoptosis induced by galectin-8. [electronic resource]
Producer: 20050712Description: 19105-14 p. digitalISSN:- 0021-9258
- Androstadienes -- pharmacology
- Animals
- Anthracenes -- pharmacology
- Apoptosis
- Blotting, Northern
- Blotting, Western
- Bromodeoxyuridine -- pharmacology
- CHO Cells
- Cell Adhesion
- Cell Cycle Proteins -- metabolism
- Cell Line, Tumor
- Cell Proliferation
- Cricetinae
- Cyclin-Dependent Kinase Inhibitor p21
- Cycloheximide -- pharmacology
- DNA -- metabolism
- Galectins -- metabolism
- Green Fluorescent Proteins -- metabolism
- Humans
- JNK Mitogen-Activated Protein Kinases -- metabolism
- Ligands
- MAP Kinase Kinase 4
- Mitogen-Activated Protein Kinase Kinases -- metabolism
- Phosphatidylinositol 3-Kinases -- metabolism
- Poly(ADP-ribose) Polymerases -- metabolism
- Protein Kinase Inhibitors -- metabolism
- Protein Synthesis Inhibitors -- pharmacology
- RNA, Messenger -- metabolism
- Reverse Transcriptase Polymerase Chain Reaction
- Thymidine -- pharmacology
- Time Factors
- Up-Regulation
- Wortmannin
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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