CD72 polymorphisms associated with alternative splicing modify susceptibility to human systemic lupus erythematosus through epistatic interaction with FCGR2B. [electronic resource]
Producer: 20050321Description: 2907-17 p. digitalISSN:- 0964-6906
- Adult
- Alternative Splicing
- Animals
- Antigens, CD -- genetics
- Antigens, Differentiation, B-Lymphocyte -- genetics
- Base Sequence
- COS Cells
- Cohort Studies
- DNA Primers
- Epistasis, Genetic
- Female
- Genetic Predisposition to Disease
- Genotype
- Humans
- Introns
- Male
- Middle Aged
- Polymorphism, Genetic
- Receptors, IgG -- genetics
- Reverse Transcriptase Polymerase Chain Reaction
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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