CD72 polymorphisms associated with alternative splicing modify susceptibility to human systemic lupus erythematosus through epistatic interaction with FCGR2B.

Hitomi, Yuki

CD72 polymorphisms associated with alternative splicing modify susceptibility to human systemic lupus erythematosus through epistatic interaction with FCGR2B. [electronic resource] - Human molecular genetics Dec 2004 - 2907-17 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't

0964-6906

10.1093/hmg/ddh318 doi


Adult
Alternative Splicing
Animals
Antigens, CD--genetics
Antigens, Differentiation, B-Lymphocyte--genetics
Base Sequence
COS Cells
Cohort Studies
DNA Primers
Epistasis, Genetic
Female
Genetic Predisposition to Disease
Genotype
Humans
Introns
Male
Middle Aged
Polymorphism, Genetic
Receptors, IgG--genetics
Reverse Transcriptase Polymerase Chain Reaction