Reduction of calcium release from the endoplasmic reticulum could only provide partial neuroprotection against beta-amyloid peptide toxicity. [electronic resource]
Producer: 20040126Description: 1413-26 p. digitalISSN:- 0022-3042
- Amyloid beta-Peptides -- toxicity
- Analysis of Variance
- Animals
- Blotting, Western
- Boron Compounds -- pharmacology
- Calcium -- metabolism
- Carrier Proteins -- metabolism
- Caspase 3
- Caspases -- metabolism
- Cell Death -- drug effects
- Cells, Cultured
- Cerebral Cortex -- cytology
- Dose-Response Relationship, Drug
- Drug Interactions
- Embryo, Mammalian
- Endoplasmic Reticulum -- drug effects
- Endoplasmic Reticulum Chaperone BiP
- Enzyme Inhibitors -- pharmacology
- Heat-Shock Proteins
- Immunosuppressive Agents -- pharmacology
- Indoles -- metabolism
- Intracellular Space -- drug effects
- L-Lactate Dehydrogenase -- metabolism
- Macrocyclic Compounds
- Molecular Chaperones -- metabolism
- Neurons -- drug effects
- Oxazoles -- pharmacology
- Peptide Fragments -- toxicity
- Rats
- Rats, Sprague-Dawley
- Tacrolimus -- pharmacology
- Time Factors
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Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
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