Akt activation disrupts mammary acinar architecture and enhances proliferation in an mTOR-dependent manner. [electronic resource]
Producer: 20031203Description: 315-26 p. digitalISSN:- 0021-9525
- Antibiotics, Antineoplastic -- pharmacology
- Apoptosis
- Breast -- cytology
- Cell Division -- drug effects
- Cell Line
- Cell Line, Transformed
- Cell Polarity -- drug effects
- Cell Size -- drug effects
- Cell Survival -- drug effects
- Cyclin D1 -- metabolism
- DNA -- analysis
- Dose-Response Relationship, Drug
- Enzyme Activation
- Epithelial Cells -- cytology
- Humans
- Morphogenesis -- drug effects
- Protein Kinase Inhibitors
- Protein Kinases -- drug effects
- Protein Serine-Threonine Kinases
- Proto-Oncogene Proteins -- genetics
- Proto-Oncogene Proteins c-akt
- Retroviridae -- genetics
- Sirolimus -- pharmacology
- TOR Serine-Threonine Kinases
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, U.S. Gov't, P.H.S.
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