Presenilin 1 facilitates the constitutive turnover of beta-catenin: differential activity of Alzheimer's disease-linked PS1 mutants in the beta-catenin-signaling pathway. [electronic resource]
Producer: 19990616Description: 4229-37 p. digitalISSN:- 1529-2401
- Adenomatous Polyposis Coli -- metabolism
- Alzheimer Disease -- genetics
- Animals
- Brain -- metabolism
- Cadherins -- physiology
- Calcium-Calmodulin-Dependent Protein Kinases -- metabolism
- Cell Line
- Cytoskeletal Proteins -- metabolism
- Glycogen Synthase Kinase 3
- Glycogen Synthase Kinases
- Membrane Proteins -- genetics
- Mice
- Mice, Transgenic
- Mutation
- Presenilin-1
- Signal Transduction -- physiology
- Trans-Activators
- beta Catenin
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Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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