000 01720 a2200457 4500
005 20250513152956.0
264 0 _c19981106
008 199811s 0 0 eng d
022 _a0021-9258
024 7 _a10.1074/jbc.273.42.27231
_2doi
040 _aNLM
_beng
_cNLM
100 1 _aKupershmidt, S
245 0 0 _aA K+ channel splice variant common in human heart lacks a C-terminal domain required for expression of rapidly activating delayed rectifier current.
_h[electronic resource]
260 _bThe Journal of biological chemistry
_cOct 1998
300 _a27231-5 p.
_bdigital
500 _aPublication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
650 0 4 _aAlternative Splicing
650 0 4 _aCation Transport Proteins
650 0 4 _aCloning, Molecular
650 0 4 _aDNA-Binding Proteins
650 0 4 _aERG1 Potassium Channel
650 0 4 _aElectric Conductivity
650 0 4 _aEther-A-Go-Go Potassium Channels
650 0 4 _aHumans
650 0 4 _aLong QT Syndrome
_xmetabolism
650 0 4 _aMolecular Sequence Data
650 0 4 _aMyocardium
_xmetabolism
650 0 4 _aPotassium
_xmetabolism
650 0 4 _aPotassium Channels
_xgenetics
650 0 4 _aPotassium Channels, Voltage-Gated
650 0 4 _aRecombinant Proteins
_xmetabolism
650 0 4 _aSequence Analysis, DNA
650 0 4 _aSequence Deletion
650 0 4 _aTrans-Activators
650 0 4 _aTranscriptional Regulator ERG
700 1 _aSnyders, D J
700 1 _aRaes, A
700 1 _aRoden, D M
773 0 _tThe Journal of biological chemistry
_gvol. 273
_gno. 42
_gp. 27231-5
856 4 0 _uhttps://doi.org/10.1074/jbc.273.42.27231
_zAvailable from publisher's website
999 _c9730061
_d9730061