000 01397 a2200445 4500
005 20250513131039.0
264 0 _c19971204
008 199712s 0 0 eng d
022 _a0197-4580
024 7 _a10.1016/s0197-4580(96)00163-7
_2doi
040 _aNLM
_beng
_cNLM
100 1 _aSoong, N W
245 0 0 _aMitochondrial DNA deletions are rare in the free radical-rich retinal environment.
_h[electronic resource]
260 _bNeurobiology of aging
_c
300 _a827-31 p.
_bdigital
500 _aPublication Type: Journal Article; Research Support, U.S. Gov't, P.H.S.
650 0 4 _aAdolescent
650 0 4 _aAdult
650 0 4 _aAged
650 0 4 _aAged, 80 and over
650 0 4 _aChild
650 0 4 _aChild, Preschool
650 0 4 _aDNA Damage
_xphysiology
650 0 4 _aDNA, Mitochondrial
_xmetabolism
650 0 4 _aFemale
650 0 4 _aFree Radicals
_xmetabolism
650 0 4 _aHumans
650 0 4 _aInfant
650 0 4 _aMale
650 0 4 _aMiddle Aged
650 0 4 _aOptic Nerve
_xmetabolism
650 0 4 _aPolymerase Chain Reaction
650 0 4 _aRetina
_xmetabolism
650 0 4 _aSequence Deletion
_xphysiology
700 1 _aDang, M H
700 1 _aHinton, D R
700 1 _aArnheim, N
773 0 _tNeurobiology of aging
_gvol. 17
_gno. 6
_gp. 827-31
856 4 0 _uhttps://doi.org/10.1016/s0197-4580(96)00163-7
_zAvailable from publisher's website
999 _c9333757
_d9333757