000			01731 a2200493 4500
005			20250513104036.0
264		0	_c19961210
008			199612s 0 0 eng d
022			_a0004-0010
024	7		_a10.1001/archsurg.1996.01430230085015 _2doi
040			_aNLM _beng _cNLM
100	1		_aMeldrum, D R
245	0	0	_aNeutrophils are required for endotoxin-induced myocardial cross-tolerance to ischemia-reperfusion injury. _h[electronic resource]
260			_bArchives of surgery (Chicago, Ill. : 1960) _cNov 1996
300			_a1203-8 p. _bdigital
500			_aPublication Type: Journal Article; Research Support, U.S. Gov't, P.H.S.
650	0	4	_aAnimals
650	0	4	_aBlood Pressure
650	0	4	_aCoronary Circulation
650	0	4	_aCreatine Kinase _xanalysis
650	0	4	_aDiastole
650	0	4	_aDisease Susceptibility _ximmunology
650	0	4	_aEndotoxins _ximmunology
650	0	4	_aHeart Arrest, Induced
650	0	4	_aLeukocyte Count
650	0	4	_aMale
650	0	4	_aMyocardial Contraction
650	0	4	_aMyocardial Ischemia _ximmunology
650	0	4	_aMyocardial Reperfusion
650	0	4	_aMyocardial Reperfusion Injury _ximmunology
650	0	4	_aMyocardium _xenzymology
650	0	4	_aNeutrophil Activation
650	0	4	_aNeutrophils _ximmunology
650	0	4	_aOxidative Stress _ximmunology
650	0	4	_aRats
650	0	4	_aRats, Sprague-Dawley
700	1		_aSheridan, B C
700	1		_aCleveland, J C
700	1		_aFullerton, D A
700	1		_aBanerjee, A
700	1		_aHarken, A H
773	0		_tArchives of surgery (Chicago, Ill. : 1960) _gvol. 131 _gno. 11 _gp. 1203-8
856	4	0	_uhttps://doi.org/10.1001/archsurg.1996.01430230085015 _zAvailable from publisher's website
999			_c8903760 _d8903760