000 01358 a2200385 4500
005 20250513103553.0
264 0 _c19961127
008 199611s 0 0 eng d
022 _a0022-1899
024 7 _a10.1093/infdis/174.5.1085
_2doi
040 _aNLM
_beng
_cNLM
100 1 _aMdluli, K
245 0 0 _aBiochemical and genetic data suggest that InhA is not the primary target for activated isoniazid in Mycobacterium tuberculosis.
_h[electronic resource]
260 _bThe Journal of infectious diseases
_cNov 1996
300 _a1085-90 p.
_bdigital
500 _aPublication Type: Journal Article
650 0 4 _aAntitubercular Agents
_xpharmacology
650 0 4 _aBacterial Proteins
_xgenetics
650 0 4 _aBiotransformation
650 0 4 _aDrug Resistance, Microbial
650 0 4 _aIsoniazid
_xmetabolism
650 0 4 _aLipids
_xbiosynthesis
650 0 4 _aMutation
650 0 4 _aMycobacterium tuberculosis
_xdrug effects
650 0 4 _aOxidoreductases
650 0 4 _aPromoter Regions, Genetic
700 1 _aSherman, D R
700 1 _aHickey, M J
700 1 _aKreiswirth, B N
700 1 _aMorris, S
700 1 _aStover, C K
700 1 _aBarry, C E
773 0 _tThe Journal of infectious diseases
_gvol. 174
_gno. 5
_gp. 1085-90
856 4 0 _uhttps://doi.org/10.1093/infdis/174.5.1085
_zAvailable from publisher's website
999 _c8889105
_d8889105