000 01266 a2200385 4500
005 20250512054934.0
264 0 _c19871130
008 198711s 0 0 eng d
022 _a0092-8674
024 7 _a10.1016/0092-8674(87)90147-4
_2doi
040 _aNLM
_beng
_cNLM
100 1 _aHonegger, A M
245 0 0 _aPoint mutation at the ATP binding site of EGF receptor abolishes protein-tyrosine kinase activity and alters cellular routing.
_h[electronic resource]
260 _bCell
_cOct 1987
300 _a199-209 p.
_bdigital
500 _aPublication Type: Journal Article
650 0 4 _aAdenosine Triphosphate
_xmetabolism
650 0 4 _aBinding Sites
650 0 4 _aCell Line
650 0 4 _aEpidermal Growth Factor
_xpharmacokinetics
650 0 4 _aErbB Receptors
_xdrug effects
650 0 4 _aHumans
650 0 4 _aMutation
650 0 4 _aProtein-Tyrosine Kinases
_xmetabolism
700 1 _aDull, T J
700 1 _aFelder, S
700 1 _aVan Obberghen, E
700 1 _aBellot, F
700 1 _aSzapary, D
700 1 _aSchmidt, A
700 1 _aUllrich, A
700 1 _aSchlessinger, J
773 0 _tCell
_gvol. 51
_gno. 2
_gp. 199-209
856 4 0 _uhttps://doi.org/10.1016/0092-8674(87)90147-4
_zAvailable from publisher's website
999 _c3504956
_d3504956