000 01723 a2200457 4500
005 20250518090046.0
264 0 _c20210119
008 202101s 0 0 eng d
022 _a1950-6007
024 7 _a10.1016/j.biopha.2019.109702
_2doi
040 _aNLM
_beng
_cNLM
100 1 _aCheng, Xiaoli
245 0 0 _a18α-Glycyrrhetinic acid (GA) ameliorates fructose-induced nephropathy in mice by suppressing oxidative stress, dyslipidemia and inflammation.
_h[electronic resource]
260 _bBiomedicine & pharmacotherapy = Biomedecine & pharmacotherapie
_cMay 2020
300 _a109702 p.
_bdigital
500 _aPublication Type: Journal Article
650 0 4 _aAcute Kidney Injury
_xchemically induced
650 0 4 _aAnimals
650 0 4 _aAnti-Inflammatory Agents
_xpharmacology
650 0 4 _aCell Survival
_xdrug effects
650 0 4 _aCytokines
_xmetabolism
650 0 4 _aDyslipidemias
_xmetabolism
650 0 4 _aFatty Acids
_xmetabolism
650 0 4 _aFructose
_xpharmacology
650 0 4 _aGlycyrrhetinic Acid
_xanalogs & derivatives
650 0 4 _aGlycyrrhiza
_xchemistry
650 0 4 _aHumans
650 0 4 _aInflammation
_xmetabolism
650 0 4 _aMAP Kinase Signaling System
_xdrug effects
650 0 4 _aMale
650 0 4 _aMice
650 0 4 _aMice, Inbred C57BL
650 0 4 _aMitochondria
_xpathology
650 0 4 _aNF-E2-Related Factor 2
_xmetabolism
650 0 4 _aNF-kappa B
_xmetabolism
650 0 4 _aOxidative Stress
_xdrug effects
700 1 _aQiu, Linwei
700 1 _aWang, Fen
773 0 _tBiomedicine & pharmacotherapy = Biomedecine & pharmacotherapie
_gvol. 125
_gp. 109702
856 4 0 _uhttps://doi.org/10.1016/j.biopha.2019.109702
_zAvailable from publisher's website
999 _c30680901
_d30680901