000			01955 a2200517 4500
005			20250518053657.0
264		0	_c20210106
008			202101s 0 0 eng d
022			_a1873-7064
024	7		_a10.1016/j.neuropharm.2019.107736 _2doi
040			_aNLM _beng _cNLM
100	1		_aWu, Han-Fang
245	0	0	_aTargeting the inhibition of fatty acid amide hydrolase ameliorate the endocannabinoid-mediated synaptic dysfunction in a valproic acid-induced rat model of Autism. _h[electronic resource]
260			_bNeuropharmacology _c01 2020
300			_a107736 p. _bdigital
500			_aPublication Type: Journal Article; Research Support, Non-U.S. Gov't
650	0	4	_aAmidohydrolases _xantagonists & inhibitors
650	0	4	_aAnimals
650	0	4	_aArachidonic Acids _xmetabolism
650	0	4	_aAutism Spectrum Disorder _xchemically induced
650	0	4	_aBenzamides _xpharmacology
650	0	4	_aCarbamates _xpharmacology
650	0	4	_aDisease Models, Animal
650	0	4	_aEndocannabinoids _xmetabolism
650	0	4	_aEnzyme Inhibitors _xtoxicity
650	0	4	_aFemale
650	0	4	_aLong-Term Synaptic Depression _xdrug effects
650	0	4	_aNeuronal Plasticity _xdrug effects
650	0	4	_aPolyunsaturated Alkamides _xmetabolism
650	0	4	_aPrefrontal Cortex _xmetabolism
650	0	4	_aPregnancy
650	0	4	_aPrenatal Exposure Delayed Effects _xchemically induced
650	0	4	_aProtein Transport _xdrug effects
650	0	4	_aRats
650	0	4	_aReceptors, AMPA _xdrug effects
650	0	4	_aReceptors, Metabotropic Glutamate _xdrug effects
650	0	4	_aSocial Behavior
650	0	4	_aValproic Acid _xtoxicity
700	1		_aLu, Ting-Yi
700	1		_aChu, Ming-Chia
700	1		_aChen, Po See
700	1		_aLee, Chi-Wei
700	1		_aLin, Hui-Ching
773	0		_tNeuropharmacology _gvol. 162 _gp. 107736
856	4	0	_uhttps://doi.org/10.1016/j.neuropharm.2019.107736 _zAvailable from publisher's website
999			_c29991535 _d29991535