000 01787 a2200493 4500
005 20250518002525.0
264 0 _c20190215
008 201902s 0 0 eng d
022 _a1553-7404
024 7 _a10.1371/journal.pgen.1007681
_2doi
040 _aNLM
_beng
_cNLM
100 1 _aIyer, Dharini
245 0 0 _aCoronary artery disease genes SMAD3 and TCF21 promote opposing interactive genetic programs that regulate smooth muscle cell differentiation and disease risk.
_h[electronic resource]
260 _bPLoS genetics
_c10 2018
300 _ae1007681 p.
_bdigital
500 _aPublication Type: Journal Article; Research Support, N.I.H., Extramural
650 0 4 _aBasic Helix-Loop-Helix Transcription Factors
_xgenetics
650 0 4 _aBinding Sites
650 0 4 _aCell Differentiation
_xgenetics
650 0 4 _aCoronary Artery Disease
_xgenetics
650 0 4 _aEpistasis, Genetic
650 0 4 _aGenetic Predisposition to Disease
650 0 4 _aGenome-Wide Association Study
650 0 4 _aHumans
650 0 4 _aMuscle, Smooth, Vascular
_xmetabolism
650 0 4 _aPolymorphism, Single Nucleotide
650 0 4 _aPrimary Cell Culture
650 0 4 _aSignal Transduction
650 0 4 _aSmad3 Protein
_xgenetics
650 0 4 _aTransforming Growth Factor beta
_xgenetics
700 1 _aZhao, Quanyi
700 1 _aWirka, Robert
700 1 _aNaravane, Ameay
700 1 _aNguyen, Trieu
700 1 _aLiu, Boxiang
700 1 _aNagao, Manabu
700 1 _aCheng, Paul
700 1 _aMiller, Clint L
700 1 _aKim, Juyong Brian
700 1 _aPjanic, Milos
700 1 _aQuertermous, Thomas
773 0 _tPLoS genetics
_gvol. 14
_gno. 10
_gp. e1007681
856 4 0 _uhttps://doi.org/10.1371/journal.pgen.1007681
_zAvailable from publisher's website
999 _c28926114
_d28926114