000 01625 a2200421 4500
005 20250517160748.0
264 0 _c20170925
008 201709s 0 0 eng d
022 _a1522-1490
024 7 _a10.1152/ajpregu.00413.2016
_2doi
040 _aNLM
_beng
_cNLM
100 1 _aRoberts, Brandon L
245 0 0 _aHigh glucose increases action potential firing of catecholamine neurons in the nucleus of the solitary tract by increasing spontaneous glutamate inputs.
_h[electronic resource]
260 _bAmerican journal of physiology. Regulatory, integrative and comparative physiology
_cSep 2017
300 _aR229-R239 p.
_bdigital
500 _aPublication Type: Journal Article
650 0 4 _aAction Potentials
_xphysiology
650 0 4 _aAnimals
650 0 4 _aCatecholamines
_xmetabolism
650 0 4 _aCells, Cultured
650 0 4 _aExcitatory Postsynaptic Potentials
_xphysiology
650 0 4 _aFemale
650 0 4 _aGlucose
_xmetabolism
650 0 4 _aGlutamic Acid
_xphysiology
650 0 4 _aMice
650 0 4 _aMice, Inbred C57BL
650 0 4 _aMice, Transgenic
650 0 4 _aNeurons
_xphysiology
650 0 4 _aNeurotransmitter Agents
_xmetabolism
650 0 4 _aSolitary Nucleus
_xcytology
650 0 4 _aSynaptic Transmission
_xphysiology
700 1 _aZhu, Mingyan
700 1 _aZhao, Huan
700 1 _aDillon, Crystal
700 1 _aAppleyard, Suzanne M
773 0 _tAmerican journal of physiology. Regulatory, integrative and comparative physiology
_gvol. 313
_gno. 3
_gp. R229-R239
856 4 0 _uhttps://doi.org/10.1152/ajpregu.00413.2016
_zAvailable from publisher's website
999 _c27278063
_d27278063