000 01788 a2200553 4500
005 20250517151605.0
264 0 _c20170926
008 201709s 0 0 eng d
022 _a1558-8238
024 7 _a10.1172/JCI88725
_2doi
040 _aNLM
_beng
_cNLM
100 1 _aYuan, Xuejun
245 0 0 _aDisruption of spatiotemporal hypoxic signaling causes congenital heart disease in mice.
_h[electronic resource]
260 _bThe Journal of clinical investigation
_cJun 2017
300 _a2235-2248 p.
_bdigital
500 _aPublication Type: Journal Article
650 0 4 _aAnimals
650 0 4 _aCell Hypoxia
650 0 4 _aCell Proliferation
650 0 4 _aEmbryo, Mammalian
_xpathology
650 0 4 _aFemale
650 0 4 _aGene Expression
650 0 4 _aGene Expression Regulation, Developmental
650 0 4 _aGene Silencing
650 0 4 _aHEK293 Cells
650 0 4 _aHeart Defects, Congenital
_xetiology
650 0 4 _aHistone Deacetylases
_xmetabolism
650 0 4 _aHomeobox Protein Nkx-2.5
_xgenetics
650 0 4 _aHumans
650 0 4 _aLIM-Homeodomain Proteins
_xgenetics
650 0 4 _aMice, Inbred C57BL
650 0 4 _aMice, Transgenic
650 0 4 _aMyocardium
_xmetabolism
650 0 4 _aPregnancy
650 0 4 _aSignal Transduction
650 0 4 _aSirtuin 1
_xgenetics
650 0 4 _aSpatio-Temporal Analysis
650 0 4 _aTranscription Factors
_xgenetics
700 1 _aQi, Hui
700 1 _aLi, Xiang
700 1 _aWu, Fan
700 1 _aFang, Jian
700 1 _aBober, Eva
700 1 _aDobreva, Gergana
700 1 _aZhou, Yonggang
700 1 _aBraun, Thomas
773 0 _tThe Journal of clinical investigation
_gvol. 127
_gno. 6
_gp. 2235-2248
856 4 0 _uhttps://doi.org/10.1172/JCI88725
_zAvailable from publisher's website
999 _c27107634
_d27107634