000 01392 a2200397 4500
005 20250517022410.0
264 0 _c20151204
008 201512s 0 0 eng d
022 _a1540-7748
024 7 _a10.1085/jgp.201411348
_2doi
040 _aNLM
_beng
_cNLM
100 1 _aAmjad, Asma
245 0 0 _aConditional knockout of TMEM16A/anoctamin1 abolishes the calcium-activated chloride current in mouse vomeronasal sensory neurons.
_h[electronic resource]
260 _bThe Journal of general physiology
_cApr 2015
300 _a285-301 p.
_bdigital
500 _aPublication Type: Journal Article; Research Support, Non-U.S. Gov't
650 0 4 _aAction Potentials
650 0 4 _aAnimals
650 0 4 _aAnoctamin-1
650 0 4 _aCalcium
_xmetabolism
650 0 4 _aCells, Cultured
650 0 4 _aChloride Channels
_xgenetics
650 0 4 _aGene Deletion
650 0 4 _aMice
650 0 4 _aSensory Receptor Cells
_xmetabolism
650 0 4 _aVomeronasal Organ
_xcytology
700 1 _aHernandez-Clavijo, Andres
700 1 _aPifferi, Simone
700 1 _aMaurya, Devendra Kumar
700 1 _aBoccaccio, Anna
700 1 _aFranzot, Jessica
700 1 _aRock, Jason
700 1 _aMenini, Anna
773 0 _tThe Journal of general physiology
_gvol. 145
_gno. 4
_gp. 285-301
856 4 0 _uhttps://doi.org/10.1085/jgp.201411348
_zAvailable from publisher's website
999 _c24714847
_d24714847