000 01629 a2200481 4500
005 20250517021745.0
264 0 _c20160120
008 201601s 0 0 eng d
022 _a2158-3188
024 7 _a10.1038/tp.2014.147
_2doi
040 _aNLM
_beng
_cNLM
100 1 _aStansfield, K H
245 0 0 _aEarly-life lead exposure recapitulates the selective loss of parvalbumin-positive GABAergic interneurons and subcortical dopamine system hyperactivity present in schizophrenia.
_h[electronic resource]
260 _bTranslational psychiatry
_cMar 2015
300 _ae522 p.
_bdigital
500 _aPublication Type: Journal Article; Research Support, N.I.H., Extramural
650 0 4 _aAnimals
650 0 4 _aBlotting, Western
650 0 4 _aBrain
_xmetabolism
650 0 4 _aChromatography, High Pressure Liquid
650 0 4 _aDopamine
_xblood
650 0 4 _aFemale
650 0 4 _aHyperkinesis
_xblood
650 0 4 _aInterneurons
_xmetabolism
650 0 4 _aLead
_xadverse effects
650 0 4 _aLead Poisoning
_xblood
650 0 4 _aMale
650 0 4 _aParvalbumins
_xblood
650 0 4 _aPregnancy
650 0 4 _aPrenatal Exposure Delayed Effects
_xblood
650 0 4 _aRats
650 0 4 _aRats, Long-Evans
650 0 4 _aRisk Factors
650 0 4 _aSchizophrenia
_xblood
650 0 4 _agamma-Aminobutyric Acid
_xblood
700 1 _aRuby, K N
700 1 _aSoares, B D
700 1 _aMcGlothan, J L
700 1 _aLiu, X
700 1 _aGuilarte, T R
773 0 _tTranslational psychiatry
_gvol. 5
_gp. e522
856 4 0 _uhttps://doi.org/10.1038/tp.2014.147
_zAvailable from publisher's website
999 _c24694143
_d24694143