000 01676 a2200445 4500
005 20250517013448.0
264 0 _c20151201
008 201512s 0 0 eng d
022 _a1873-7064
024 7 _a10.1016/j.neuropharm.2015.01.007
_2doi
040 _aNLM
_beng
_cNLM
100 1 _aZhang, Xiaoqun
245 0 0 _aDopamine depletion of the striatum causes a cell-type specific reorganization of GluN2B- and GluN2D-containing NMDA receptors.
_h[electronic resource]
260 _bNeuropharmacology
_cMay 2015
300 _a108-15 p.
_bdigital
500 _aPublication Type: Journal Article; Research Support, Non-U.S. Gov't
650 0 4 _aAdrenergic Agents
_xtoxicity
650 0 4 _aAnimals
650 0 4 _aAntioxidants
_xtoxicity
650 0 4 _aAscorbic Acid
_xtoxicity
650 0 4 _aBrain Injuries
_xchemically induced
650 0 4 _aCorpus Striatum
_xcytology
650 0 4 _aDisease Models, Animal
650 0 4 _aDopamine
_xdeficiency
650 0 4 _aDose-Response Relationship, Drug
650 0 4 _aExcitatory Amino Acid Agents
_xpharmacology
650 0 4 _aExcitatory Postsynaptic Potentials
_xdrug effects
650 0 4 _aGene Expression Regulation
_xdrug effects
650 0 4 _aMale
650 0 4 _aMice
650 0 4 _aMice, Inbred C57BL
650 0 4 _aNeurons
_xdrug effects
650 0 4 _aOxidopamine
_xtoxicity
650 0 4 _aPatch-Clamp Techniques
650 0 4 _aReceptors, N-Methyl-D-Aspartate
_xmetabolism
650 0 4 _aTyrosine 3-Monooxygenase
_xmetabolism
700 1 _aChergui, Karima
773 0 _tNeuropharmacology
_gvol. 92
_gp. 108-15
856 4 0 _uhttps://doi.org/10.1016/j.neuropharm.2015.01.007
_zAvailable from publisher's website
999 _c24563846
_d24563846