000 01726 a2200505 4500
005 20250516214859.0
264 0 _c20140905
008 201409s 0 0 eng d
022 _a1479-6805
024 7 _a10.1530/JOE-14-0161
_2doi
040 _aNLM
_beng
_cNLM
100 1 _aOgata, Hidetada
245 0 0 _aKATP channel as well as SGLT1 participates in GIP secretion in the diabetic state.
_h[electronic resource]
260 _bThe Journal of endocrinology
_cAug 2014
300 _a191-200 p.
_bdigital
500 _aPublication Type: Journal Article; Research Support, Non-U.S. Gov't
650 0 4 _aAnimals
650 0 4 _aDiabetes Mellitus, Experimental
_xphysiopathology
650 0 4 _aDiazoxide
_xpharmacology
650 0 4 _aGastric Inhibitory Polypeptide
_xmetabolism
650 0 4 _aGlucose
_xpharmacology
650 0 4 _aKATP Channels
_xphysiology
650 0 4 _aMice
650 0 4 _aPhlorhizin
_xpharmacology
650 0 4 _aPotassium Channels, Inwardly Rectifying
_xdeficiency
650 0 4 _aSodium-Glucose Transporter 1
_xphysiology
650 0 4 _aSulfonylurea Compounds
_xpharmacology
700 1 _aSeino, Yusuke
700 1 _aHarada, Norio
700 1 _aIida, Atsushi
700 1 _aSuzuki, Kazuyo
700 1 _aIzumoto, Takako
700 1 _aIshikawa, Kota
700 1 _aUenishi, Eita
700 1 _aOzaki, Nobuaki
700 1 _aHayashi, Yoshitaka
700 1 _aMiki, Takashi
700 1 _aInagaki, Nobuya
700 1 _aTsunekawa, Shin
700 1 _aHamada, Yoji
700 1 _aSeino, Susumu
700 1 _aOiso, Yutaka
773 0 _tThe Journal of endocrinology
_gvol. 222
_gno. 2
_gp. 191-200
856 4 0 _uhttps://doi.org/10.1530/JOE-14-0161
_zAvailable from publisher's website
999 _c23882645
_d23882645