000 01422 a2200373 4500
005 20250516084807.0
264 0 _c20120611
008 201206s 0 0 eng d
022 _a1550-6606
024 7 _a10.4049/jimmunol.1200021
_2doi
040 _aNLM
_beng
_cNLM
100 1 _aHäcker, Hans
245 0 0 _aNIK prevents the development of hypereosinophilic syndrome-like disease in mice independent of IKKα activation.
_h[electronic resource]
260 _bJournal of immunology (Baltimore, Md. : 1950)
_cMay 2012
300 _a4602-10 p.
_bdigital
500 _aPublication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
650 0 4 _aAnimals
650 0 4 _aEnzyme Activation
_xgenetics
650 0 4 _aHypereosinophilic Syndrome
_xgenetics
650 0 4 _aI-kappa B Kinase
_xgenetics
650 0 4 _aInflammation
_xgenetics
650 0 4 _aMice
650 0 4 _aMice, Knockout
650 0 4 _aMutation
650 0 4 _aProtein Serine-Threonine Kinases
_xgenetics
650 0 4 _aProtein Structure, Secondary
650 0 4 _aTh2 Cells
_ximmunology
650 0 4 _aNF-kappaB-Inducing Kinase
700 1 _aChi, Liying
700 1 _aRehg, Jerold E
700 1 _aRedecke, Vanessa
773 0 _tJournal of immunology (Baltimore, Md. : 1950)
_gvol. 188
_gno. 9
_gp. 4602-10
856 4 0 _uhttps://doi.org/10.4049/jimmunol.1200021
_zAvailable from publisher's website
999 _c21676305
_d21676305