000			01744 a2200457 4500
005			20250516065821.0
264		0	_c20120325
008			201203s 0 0 eng d
022			_a1525-2191
024	7		_a10.1016/j.ajpath.2011.10.018 _2doi
040			_aNLM _beng _cNLM
100	1		_aSu, Wen-Hong
245	0	0	_aRadiation-induced increase in cell migration and metastatic potential of cervical cancer cells operates via the K-Ras pathway. _h[electronic resource]
260			_bThe American journal of pathology _cFeb 2012
300			_a862-71 p. _bdigital
500			_aPublication Type: Journal Article; Research Support, Non-U.S. Gov't
650	0	4	_aAnimals
650	0	4	_aCell Movement _xradiation effects
650	0	4	_aFemale
650	0	4	_aLung Neoplasms _xsecondary
650	0	4	_aMAP Kinase Signaling System _xradiation effects
650	0	4	_aMice
650	0	4	_aMice, Inbred BALB C
650	0	4	_aMice, Nude
650	0	4	_aNeoplasm Metastasis
650	0	4	_aNeoplasm Recurrence, Local _xmetabolism
650	0	4	_aNeoplasms, Radiation-Induced _xmetabolism
650	0	4	_aPhosphorylation
650	0	4	_aProto-Oncogene Proteins c-raf _xmetabolism
650	0	4	_aRadiation Tolerance _xradiation effects
650	0	4	_aTumor Cells, Cultured
650	0	4	_aUterine Cervical Neoplasms _xmetabolism
650	0	4	_aWound Healing _xradiation effects
650	0	4	_ap38 Mitogen-Activated Protein Kinases _xphysiology
650	0	4	_aras Proteins _xgenetics
700	1		_aChuang, Pei-Chin
700	1		_aHuang, Eng-Yen
700	1		_aYang, Kuender D
773	0		_tThe American journal of pathology _gvol. 180 _gno. 2 _gp. 862-71
856	4	0	_uhttps://doi.org/10.1016/j.ajpath.2011.10.018 _zAvailable from publisher's website
999			_c21363757 _d21363757