000 01687 a2200493 4500
005 20250516005900.0
264 0 _c20110617
008 201106s 0 0 eng d
022 _a1522-1598
024 7 _a10.1152/jn.00184.2010
_2doi
040 _aNLM
_beng
_cNLM
100 1 _aChen, Shmuel
245 0 0 _aAn increase in persistent sodium current contributes to intrinsic neuronal bursting after status epilepticus.
_h[electronic resource]
260 _bJournal of neurophysiology
_cJan 2011
300 _a117-29 p.
_bdigital
500 _aPublication Type: Journal Article; Research Support, Non-U.S. Gov't
650 0 4 _aAnimals
650 0 4 _aCA1 Region, Hippocampal
_xcytology
650 0 4 _aCalcium Channel Blockers
_xpharmacology
650 0 4 _aCalcium Channels, T-Type
_xdrug effects
650 0 4 _aMale
650 0 4 _aModels, Animal
650 0 4 _aNeurons
_xdrug effects
650 0 4 _aPatch-Clamp Techniques
650 0 4 _aPilocarpine
_xadverse effects
650 0 4 _aRats
650 0 4 _aRats, Inbred Strains
650 0 4 _aRats, Wistar
650 0 4 _aSodium Channel Blockers
_xpharmacology
650 0 4 _aSodium Channels
_xdrug effects
650 0 4 _aStatus Epilepticus
_xchemically induced
650 0 4 _aTetrodotoxin
_xpharmacology
650 0 4 _aTime Factors
700 1 _aSu, Hailing
700 1 _aYue, Cuiyong
700 1 _aRemy, Stefan
700 1 _aRoyeck, Michel
700 1 _aSochivko, Dmitry
700 1 _aOpitz, Thoralf
700 1 _aBeck, Heinz
700 1 _aYaari, Yoel
773 0 _tJournal of neurophysiology
_gvol. 105
_gno. 1
_gp. 117-29
856 4 0 _uhttps://doi.org/10.1152/jn.00184.2010
_zAvailable from publisher's website
999 _c20281126
_d20281126