000 01098 a2200301 4500
005 20250515223216.0
264 0 _c20100907
008 201009s 0 0 eng d
022 _a1878-4186
024 7 _a10.1016/j.str.2010.02.012
_2doi
040 _aNLM
_beng
_cNLM
100 1 _aPeterson, Francis C
245 0 0 _aA single mutation promotes amyloidogenicity through a highly promiscuous dimer interface.
_h[electronic resource]
260 _bStructure (London, England : 1993)
_cMay 2010
300 _a563-70 p.
_bdigital
500 _aPublication Type: Journal Article; Research Support, N.I.H., Extramural
650 0 4 _aAmyloid
_xchemistry
650 0 4 _aAmyloidosis
_xgenetics
650 0 4 _aHumans
650 0 4 _aMutation
650 0 4 _aProteostasis Deficiencies
700 1 _aBaden, Elizabeth M
700 1 _aOwen, Barbara A L
700 1 _aVolkman, Brian F
700 1 _aRamirez-Alvarado, Marina
773 0 _tStructure (London, England : 1993)
_gvol. 18
_gno. 5
_gp. 563-70
856 4 0 _uhttps://doi.org/10.1016/j.str.2010.02.012
_zAvailable from publisher's website
999 _c19817285
_d19817285