000 01373 a2200385 4500
005 20250515173158.0
264 0 _c20110621
008 201106s 0 0 eng d
022 _a1573-6903
024 7 _a10.1007/s11064-009-0007-8
_2doi
040 _aNLM
_beng
_cNLM
100 1 _aMa, Guozhao
245 0 0 _aDiazoxide reverses the enhanced expression of KATP subunits in cholinergic neurons caused by exposure to Aβ₁₋₄₂.
_h[electronic resource]
260 _bNeurochemical research
_cDec 2009
300 _a2133-40 p.
_bdigital
500 _aPublication Type: Journal Article; Research Support, Non-U.S. Gov't
650 0 4 _aAlzheimer Disease
_xmetabolism
650 0 4 _aAmyloid beta-Peptides
_xpharmacology
650 0 4 _aAnimals
650 0 4 _aCholinergic Agents
_xpharmacology
650 0 4 _aDiazoxide
_xpharmacology
650 0 4 _aKATP Channels
_xbiosynthesis
650 0 4 _aNeurons
_xdrug effects
650 0 4 _aPeptide Fragments
_xpharmacology
650 0 4 _aProsencephalon
_xdrug effects
650 0 4 _aRats
700 1 _aGao, Jianxin
700 1 _aFu, Qingxi
700 1 _aJiang, Liangliang
700 1 _aWang, Ruixia
700 1 _aZhang, Yong
700 1 _aLiu, Kejing
773 0 _tNeurochemical research
_gvol. 34
_gno. 12
_gp. 2133-40
856 4 0 _uhttps://doi.org/10.1007/s11064-009-0007-8
_zAvailable from publisher's website
999 _c18913605
_d18913605