000 01556 a2200409 4500
005 20250515012309.0
264 0 _c20060309
008 200603s 0 0 eng d
022 _a0022-1767
024 7 _a10.4049/jimmunol.176.2.1259
_2doi
040 _aNLM
_beng
_cNLM
100 1 _aYu, Shiguang
245 0 0 _aThyrocytes responding to IFN-gamma are essential for development of lymphocytic spontaneous autoimmune thyroiditis and inhibition of thyrocyte hyperplasia.
_h[electronic resource]
260 _bJournal of immunology (Baltimore, Md. : 1950)
_cJan 2006
300 _a1259-65 p.
_bdigital
500 _aPublication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
650 0 4 _aAdoptive Transfer
650 0 4 _aAnimals
650 0 4 _aBase Sequence
650 0 4 _aChemokines
_xgenetics
650 0 4 _aHyperplasia
650 0 4 _aInterferon-gamma
_xdeficiency
650 0 4 _aLymphocytes
_ximmunology
650 0 4 _aMale
650 0 4 _aMice
650 0 4 _aMice, Inbred NOD
650 0 4 _aMice, Knockout
650 0 4 _aRNA, Messenger
_xgenetics
650 0 4 _aReceptors, Interferon
_xdeficiency
650 0 4 _aThyroid Gland
_ximmunology
650 0 4 _aThyroiditis, Autoimmune
_xetiology
650 0 4 _aInterferon gamma Receptor
700 1 _aSharp, Gordon C
700 1 _aBraley-Mullen, Helen
773 0 _tJournal of immunology (Baltimore, Md. : 1950)
_gvol. 176
_gno. 2
_gp. 1259-65
856 4 0 _uhttps://doi.org/10.4049/jimmunol.176.2.1259
_zAvailable from publisher's website
999 _c16011791
_d16011791