000 01717 a2200457 4500
005 20250515010741.0
264 0 _c20060322
008 200603s 0 0 eng d
022 _a0022-1767
024 7 _a10.4049/jimmunol.175.12.8154
_2doi
040 _aNLM
_beng
_cNLM
100 1 _aCheung, Yui-Ho
245 0 0 _aFunctional interplay between intrinsic B and T cell defects leads to amplification of autoimmune disease in New Zealand black chromosome 1 congenic mice.
_h[electronic resource]
260 _bJournal of immunology (Baltimore, Md. : 1950)
_cDec 2005
300 _a8154-64 p.
_bdigital
500 _aPublication Type: Journal Article; Research Support, Non-U.S. Gov't
650 0 4 _aAnimals
650 0 4 _aAntibodies, Antinuclear
_xbiosynthesis
650 0 4 _aAntigen-Presenting Cells
_ximmunology
650 0 4 _aAutoimmune Diseases
_ximmunology
650 0 4 _aAutoimmunity
_xgenetics
650 0 4 _aB-Lymphocytes
_ximmunology
650 0 4 _aCD4-Positive T-Lymphocytes
_ximmunology
650 0 4 _aCell Communication
_ximmunology
650 0 4 _aCell Proliferation
650 0 4 _aCells, Cultured
650 0 4 _aChromosomes, Mammalian
650 0 4 _aImmunoglobulin Class Switching
650 0 4 _aLymphocyte Activation
650 0 4 _aMice
650 0 4 _aMice, Congenic
650 0 4 _aMice, Inbred NZB
650 0 4 _aT-Lymphocytes
_ximmunology
700 1 _aChang, Nan-Hua
700 1 _aCai, Yong-Chun
700 1 _aBonventi, Gabriel
700 1 _aMacLeod, Ralph
700 1 _aWither, Joan E
773 0 _tJournal of immunology (Baltimore, Md. : 1950)
_gvol. 175
_gno. 12
_gp. 8154-64
856 4 0 _uhttps://doi.org/10.4049/jimmunol.175.12.8154
_zAvailable from publisher's website
999 _c15960644
_d15960644