000 01853 a2200469 4500
005 20250514201841.0
264 0 _c20041115
008 200411s 0 0 eng d
022 _a0022-1767
024 7 _a10.4049/jimmunol.173.7.4708
_2doi
040 _aNLM
_beng
_cNLM
100 1 _aBoos, Laura
245 0 0 _aDeletion of the complement anaphylatoxin C3a receptor attenuates, whereas ectopic expression of C3a in the brain exacerbates, experimental autoimmune encephalomyelitis.
_h[electronic resource]
260 _bJournal of immunology (Baltimore, Md. : 1950)
_cOct 2004
300 _a4708-14 p.
_bdigital
500 _aPublication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
650 0 4 _aAnimals
650 0 4 _aBrain
_ximmunology
650 0 4 _aCell Movement
_xgenetics
650 0 4 _aCells, Cultured
650 0 4 _aComplement C3a
_xbiosynthesis
650 0 4 _aEncephalomyelitis, Autoimmune, Experimental
_xgenetics
650 0 4 _aGene Deletion
650 0 4 _aGlial Fibrillary Acidic Protein
_xgenetics
650 0 4 _aGlycoproteins
_xadministration & dosage
650 0 4 _aHumans
650 0 4 _aMembrane Proteins
_xbiosynthesis
650 0 4 _aMice
650 0 4 _aMice, Inbred C57BL
650 0 4 _aMice, Knockout
650 0 4 _aMice, Transgenic
650 0 4 _aMyelin Sheath
_xpathology
650 0 4 _aMyelin-Oligodendrocyte Glycoprotein
650 0 4 _aPeptide Fragments
_xadministration & dosage
650 0 4 _aReceptors, Complement
_xbiosynthesis
700 1 _aCampbell, Iain L
700 1 _aAmes, Robert
700 1 _aWetsel, Rick A
700 1 _aBarnum, Scott R
773 0 _tJournal of immunology (Baltimore, Md. : 1950)
_gvol. 173
_gno. 7
_gp. 4708-14
856 4 0 _uhttps://doi.org/10.4049/jimmunol.173.7.4708
_zAvailable from publisher's website
999 _c15076738
_d15076738