000 | 01870 a2200505 4500 | ||
---|---|---|---|
005 | 20250514200538.0 | ||
264 | 0 | _c20050201 | |
008 | 200502s 0 0 eng d | ||
022 | _a0028-1298 | ||
024 | 7 |
_a10.1007/s00210-004-0955-0 _2doi |
|
040 |
_aNLM _beng _cNLM |
||
100 | 1 | _avan den Hoff, Maurice J B | |
245 | 0 | 0 |
_aIncreased cardiac workload by closure of the ductus arteriosus leads to hypertrophy and apoptosis rather than to hyperplasia in the late fetal period. _h[electronic resource] |
260 |
_bNaunyn-Schmiedeberg's archives of pharmacology _cSep 2004 |
||
300 |
_a193-202 p. _bdigital |
||
500 | _aPublication Type: Journal Article; Research Support, Non-U.S. Gov't | ||
650 | 0 | 4 | _aAnimals |
650 | 0 | 4 |
_aAnti-Inflammatory Agents, Non-Steroidal _xtoxicity |
650 | 0 | 4 |
_aAntimetabolites _xpharmacology |
650 | 0 | 4 | _aApoptosis |
650 | 0 | 4 | _aBody Weight |
650 | 0 | 4 |
_aBromodeoxyuridine _xpharmacology |
650 | 0 | 4 | _aCell Death |
650 | 0 | 4 |
_aConstriction, Pathologic _xchemically induced |
650 | 0 | 4 |
_aDuctus Arteriosus _xpathology |
650 | 0 | 4 |
_aFetal Heart _xgrowth & development |
650 | 0 | 4 | _aGene Expression Regulation, Developmental |
650 | 0 | 4 | _aHyperplasia |
650 | 0 | 4 |
_aHypertrophy _xetiology |
650 | 0 | 4 | _aIn Situ Hybridization |
650 | 0 | 4 | _aIn Situ Nick-End Labeling |
650 | 0 | 4 |
_aIndomethacin _xtoxicity |
650 | 0 | 4 | _aOrgan Size |
650 | 0 | 4 | _aRats |
650 | 0 | 4 | _aRats, Wistar |
700 | 1 | _aDeprez, Ronald H Lekanne | |
700 | 1 | _aRuijter, Jan M | |
700 | 1 | _ade Boer, Piet A J | |
700 | 1 | _aTesink-Taekema, Sabina | |
700 | 1 | _aBuffing, Anita A | |
700 | 1 | _aLamers, Wouter H | |
700 | 1 | _aMoorman, Antoon F M | |
773 | 0 |
_tNaunyn-Schmiedeberg's archives of pharmacology _gvol. 370 _gno. 3 _gp. 193-202 |
|
856 | 4 | 0 |
_uhttps://doi.org/10.1007/s00210-004-0955-0 _zAvailable from publisher's website |
999 |
_c15037869 _d15037869 |