000 01637 a2200457 4500
005 20250514183143.0
264 0 _c20040521
008 200405s 0 0 eng d
022 _a0006-291X
024 7 _a10.1016/j.bbrc.2004.02.160
_2doi
040 _aNLM
_beng
_cNLM
100 1 _aHong, Feng
245 0 0 _aTransgenic mice overexpressing cyclophilin A are resistant to cyclosporin A-induced nephrotoxicity via peptidyl-prolyl cis-trans isomerase activity.
_h[electronic resource]
260 _bBiochemical and biophysical research communications
_cApr 2004
300 _a1073-80 p.
_bdigital
500 _aPublication Type: Journal Article; Research Support, Non-U.S. Gov't
650 0 4 _aAnimals
650 0 4 _aAntioxidants
_xmetabolism
650 0 4 _aCyclophilin A
_xgenetics
650 0 4 _aCyclosporine
_xpharmacology
650 0 4 _aDose-Response Relationship, Drug
650 0 4 _aDrug Resistance
_xphysiology
650 0 4 _aKidney
_xmetabolism
650 0 4 _aMice
650 0 4 _aMice, Transgenic
650 0 4 _aPeptidylprolyl Isomerase
_xmetabolism
650 0 4 _aRecombinant Proteins
_xmetabolism
650 0 4 _aSurvival Rate
700 1 _aLee, Jinhwa
700 1 _aPiao, Yu Ji
700 1 _aJae, Yeong Kwon
700 1 _aKim, Young-Joo
700 1 _aOh, Changkyu
700 1 _aSeo, Jeong-Sun
700 1 _aYun, Yeon Sook
700 1 _aYang, Chul Woo
700 1 _aHa, Joohun
700 1 _aKim, Sung Soo
773 0 _tBiochemical and biophysical research communications
_gvol. 316
_gno. 4
_gp. 1073-80
856 4 0 _uhttps://doi.org/10.1016/j.bbrc.2004.02.160
_zAvailable from publisher's website
999 _c14761124
_d14761124