000			01919 a2200505 4500
005			20250514164411.0
264		0	_c20040402
008			200404s 0 0 eng d
022			_a0022-1767
024	7		_a10.4049/jimmunol.172.1.97 _2doi
040			_aNLM _beng _cNLM
100	1		_aWang, Wei
245	0	0	_aThe susceptibility to experimental myasthenia gravis of STAT6-/- and STAT4-/- BALB/c mice suggests a pathogenic role of Th1 cells. _h[electronic resource]
260			_bJournal of immunology (Baltimore, Md. : 1950) _cJan 2004
300			_a97-103 p. _bdigital
500			_aPublication Type: Journal Article; Research Support, U.S. Gov't, P.H.S.
650	0	4	_aAnimals
650	0	4	_aAutoantibodies _xbiosynthesis
650	0	4	_aCD4-Positive T-Lymphocytes _xcytology
650	0	4	_aCell Division _ximmunology
650	0	4	_aDNA-Binding Proteins _xdeficiency
650	0	4	_aFemale
650	0	4	_aGenetic Predisposition to Disease
650	0	4	_aImmunoglobulin G _xbiosynthesis
650	0	4	_aInterferon-gamma _xmetabolism
650	0	4	_aInterleukin-4 _xmetabolism
650	0	4	_aMice
650	0	4	_aMice, Inbred BALB C
650	0	4	_aMice, Knockout
650	0	4	_aMyasthenia Gravis, Autoimmune, Experimental _xgenetics
650	0	4	_aPeptide Fragments _xpharmacology
650	0	4	_aProtein Subunits _xpharmacology
650	0	4	_aReceptors, Nicotinic _ximmunology
650	0	4	_aSTAT4 Transcription Factor
650	0	4	_aSTAT6 Transcription Factor
650	0	4	_aSignal Transduction _xgenetics
650	0	4	_aTh1 Cells _ximmunology
650	0	4	_aTorpedo _ximmunology
650	0	4	_aTrans-Activators _xdeficiency
700	1		_aOstlie, Norma S
700	1		_aConti-Fine, Bianca M
700	1		_aMilani, Monica
773	0		_tJournal of immunology (Baltimore, Md. : 1950) _gvol. 172 _gno. 1 _gp. 97-103
856	4	0	_uhttps://doi.org/10.4049/jimmunol.172.1.97 _zAvailable from publisher's website
999			_c14419709 _d14419709